Brain Dyslexia


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Identification of potential causes for brain dyslexia:

Globally there have been widespread studies by medical scientists on the patterns of brain dyslexia to find the possible causes for this disorder. The experiments have been conducted on a control group comprising of people who are non-dyslexic and compared with brain patterns of another group comprising of dyslexics so as to pinpoint the differences.

By looking at the data from the two groups four important changes can be noticed in the brain that might indicate the early beginnings of brain dyslexia. It is of prime importance to understand these changes or the factors that have an important role to play in the future development of brain dyslexia.

Dyslexic brains have less white substance in the left parietotemporal region:

The first significant change notices while studying the brain patters of dyslexics is that these people have less white substance in the left parietotemporal region in comparison to normal individuals.

Reading skills are linked with this white substance. This explains why dyslexics stumble in their reading during their school years. Tests on the brains of dyslexics have shown that reduced white substance leads to restrictions in the ability to communicate between the various sections of the brain. This can lead to many other difficulties and problems.

In dyslexics there is no movement in V5/MT region of the brain:

There have been researches dealing with the visual parts of the brains of dyslexics and how these can cause specific disorders in individuals with brain dyslexia. The tests of moving dots have been conducted on normal as well as dyslexic groups and have produced certain definite results. The normal persons showed intensive activity in this V5/MT brain region that is connected with the visual cortex. But in the case of dyslexics with brain dyslexia no such activity was noticed in that particular region. This study through mapping of brain activity confirms how impairment of sight may lead to sending of incomplete information to the brain causing dyslexia.

Comparing the results from tests on Cerebral Blood Flow (rCBF) in certain regions of the brain:

By comparing the many activity tests on dyslexics with brain dyslexia and normal people the rCBF tests have produced results that are contrasting. For the normal group higher readings of rCBF denote better skills in reading. But for dyslexics it is the reverse where higher rCBF readings lead to worse reading powers. This can allow one to conclude that the left side of the brain of dyslexics is totally different from the normal group. The latter do much better having strong activity in the left side of the brain but this condition becomes worse for dyslexics.

Ectopias and their symmetry in the cerebral cortex:

While undertaking studies of the brain of dyslexics, ectopias (small branches of neurons and axons) are used. These control language and reading operations. They migrate to the cortex at the time of the infant’s birth. But in the instance of dyslexics it has been noticed that many of these ectopias surface in different locations away from the cortex and this leads to changes in the functions of the brain.

This proves one dyslexia theory that it is cognitive as ectopia is connected with the history of the family. The planum temporale comprises of the same size in both the hemispheres in dyslexics showing the brain of the latter is much more symmetrical in comparison to normal people.

The structure of the brain and operational differences are mainly responsible for these alterations from the norm that can cause the starting off of the first stage of brain dyslexia.

Medical science continues to study and research on the brain of dyslexics and this might lead to finding out the regions of incompatibility. But it requires the analysts to make in depth studies that will help in pinpointing the core secrets of the problem of brain dyslexia.


What is Dyslexia